Cardiology

Cellular Cardiology

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Sympathetic nerve activity has been reported to be increased in both humans and animals with chronic heart failure. One of the mechanisms believed to be responsible for this phenomenon is increased systemic and cerebral angiotensin II signaling. Plasma angiotensin II is increased in humans and animals with chronic heart failure. Blocking angiotensin II type 1 receptors decreases sympathetic nerve activity and cardiac sympathetic afferent reflex when therapy is administered to the paraventricular nucleus.. Cardiac fibroblasts (CF) are well-established as key regulators of extracellular matrix (ECM) turnover in the context of myocardial remodelling and fibrosis. The increasing availability of detailed, atrial-specific experimental data has stimulated the development of novel computational models of atrial-cellular electrophysiology and Ca²⁺ -handling. Dysregulation of autophagy in cardiomyocytes is implicated in various heart disease conditions. Beclin 1, a mammalian ortholog (Genes of different species that evolved from common ancestral gene).

Related Conferences

7^th World Cardiothoracic Meeting March 29-30, 2016 Atlanta, USA; Echo Cardiography Conference June 06-07, 2016 London, UK; Ischemic Heart Diseases Conference May 12-13, 2016 Chicago, USA; 8^th Global Cardiologists Annual Meeting July 18-20, 2016 Berlin, Germany; Annual Conference on Atherosclerosis July 11-12, 2016 Philadelphia, USA; Drug Discovery and Therapy World Congress May 11-14, 2016, Boston, USA; 7^th International Conference on Drug Discovery and Therapy, February 15-18, 2016 Dubai, UAE; Arrhythmias & the Heart: A Cardiovascular Update, February 12–16, 2016, Riyadh, Saudi Arabia; 16^th Annual International Symposium on Congenital Heart Disease, February 13–16, 2016, St. Petersburg, USA; Cardiovascular Conference at Snowbird, February 12-15, 2016, Snowbird, Utah; Korean Cardiology Related Societies Joint Scientific Congress, April 15-16, 2016, Daegu, Korea.